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Considering the potential for therapeutic development of (in)GSH, the established safety and tolerability data, biological plausibility, and pilot level clinical evidence of benefit are further supported with this demonstration that (in)GSH is able to augment brain GSH levels

Prolonged myostatin suppression can trigger compensatory upregulation of other TGF- family members (activin A, GDF-11) that partially restore growth inhibition through redundant pathways